Search results for "ventral striatum"

showing 10 items of 25 documents

Seeking the “Beauty Center” in the Brain: A Meta-Analysis of fMRI Studies of Beautiful Human Faces and Visual Art

2020

AbstractThe existence of a common beauty is a long-standing debate in philosophy and related disciplines. In the last two decades, cognitive neuroscientists have sought to elucidate this issue by exploring the common neural basis of the experience of beauty. Still, empirical evidence for such common neural basis of different forms of beauty is not conclusive. To address this question, we performed an activation likelihood estimation (ALE) meta-analysis on the existing neuroimaging studies of beauty appreciation of faces and visual art by non-expert adults (49 studies, 982 participants, meta-data are available at https://osf.io/s9xds/). We observed that perceiving these two forms of beauty a…

AdultCognitive Neurosciencemedia_common.quotation_subjectVentromedial prefrontal cortexPrefrontal Cortex050105 experimental psychologyArticleVisual arts03 medical and health sciencesBehavioral NeuroscienceBeauty0302 clinical medicineNeuroimagingmedicineHumans0501 psychology and cognitive sciencesddc:610Prefrontal cortexmedia_commonBrain Mappingmedicine.diagnostic_test05 social sciencesVentral striatumBrainCognitionActivation likelihood estimationMagnetic Resonance Imagingmedicine.anatomical_structureMeta-analysisBeautyFunctional magnetic resonance imagingPsychology030217 neurology & neurosurgery
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A ventral striatal prediction error signal in human fear extinction learning.

2021

Animal studies have shown that the prediction error (PE) signal that drives fear extinction learning is encoded by phasic activity of midbrain dopamine (DA) neurons. Thus, the extinction PE resembles the appetitive PE that drives reward learning. In humans, fear extinction learning is less well understood. Using computational neuroimaging, a previous study from our group reported hemodynamic activity in the left ventral putamen, a subregion of the ventral striatum (VS), to correlate with a PE function derived from a formal associative learning model. The activity was modulated by genetic variation in a DA-related gene. To conceptually replicate and extend this finding, we here asked whether…

AdultMaleCognitive NeuroscienceBiology050105 experimental psychologylcsh:RC321-571Extinction PsychologicalMidbrain03 medical and health sciencesYoung Adult0302 clinical medicineNeuroimagingDopaminemedicineHumansLearning0501 psychology and cognitive scienceslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryPutamen05 social sciencesVentral striatumExtinction (psychology)FearGalvanic Skin ResponseMagnetic Resonance ImagingElectric StimulationAssociative learningmedicine.anatomical_structureNeurologyVentral StriatumFemaleAnimal studiesNeuroscience030217 neurology & neurosurgeryPhotic Stimulationmedicine.drugForecastingNeuroImage
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Chronic alcohol intake abolishes the relationship between dopamine synthesis capacity and learning signals in ventral striatum

2014

Drugs of abuse elicit dopamine release in the ventral striatum, possibly biasing dopamine-driven reinforcement learning towards drug-related reward at the expense of non-drug-related reward. Indeed, in alcohol-dependent patients, reactivity in dopaminergic target areas is shifted from non-drug-related stimuli towards drug-related stimuli. Such 'hijacked' dopamine signals may impair flexible learning from non-drug-related rewards, and thus promote craving for the drug of abuse. Here, we used functional magnetic resonance imaging to measure ventral striatal activation by reward prediction errors (RPEs) during a probabilistic reversal learning task in recently detoxified alcohol-dependent pati…

AdultMaleDopamine synthesisDopamine610 Medicine & healthCravingArticle170 EthicsRewardDopaminemedicineHumansLearningDepartment Sport- und Gesundheitswissenschaften10237 Institute of Biomedical EngineeringCravingmedicine.diagnostic_testGeneral NeuroscienceDopaminergicAlcohol dependenceVentral striatum2800 General NeuroscienceMiddle AgedChronic alcoholCorpus StriatumAlcoholismmedicine.anatomical_structureCase-Control Studiesmedicine.symptomFunctional magnetic resonance imagingPsychologyNeurosciencepsychological phenomena and processesSignal Transductionmedicine.drug
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Ventral striatal prediction error signaling is associated with dopamine synthesis capacity and fluid intelligence.

2012

Fluid intelligence represents the capacity for flexible problem solving and rapid behavioral adaptation. Rewards drive flexible behavioral adaptation, in part via a teaching signal expressed as reward prediction errors in the ventral striatum, which has been associated with phasic dopamine release in animal studies. We examined a sample of 28 healthy male adults using multimodal imaging and biological parametric mapping with 1) functional magnetic resonance imaging during a reversal learning task and 2) in a subsample of 17 subjects also with positron emission tomography using 6-[18F]fluoro-L-DOPA to assess dopamine synthesis capacity. Fluid intelligence was measured using a battery of nine…

AdultMaleDopamine synthesisDopamineIntelligenceBasal GangliaArticleYoung AdultText miningDopaminemedicineHumansRadiology Nuclear Medicine and imagingProblem SolvingIntelligence TestsBrain MappingRadiological and Ultrasound TechnologyIntelligence quotientmedicine.diagnostic_testbusiness.industryVentral striatumNeuropsychologyMiddle AgedMagnetic Resonance Imagingmedicine.anatomical_structureNeurologyPositron emission tomographyPositron-Emission TomographyNeurology (clinical)AnatomyPsychologyFunctional magnetic resonance imagingbusinessNeurosciencemedicine.drugHuman brain mapping
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Net influx of plasma 6-[18F]fluoro-L-DOPA (FDOPA) to the ventral striatum correlates with prefrontal processing of affective stimuli.

2006

Dopaminergic neurotransmission in the ventral and dorsal striatum interact with central processing of rewarding and reward-indicating stimuli, and may affect frontocortical-striatal-thalamic circuits regulating goal-directed behaviour. Thirteen healthy male volunteers were investigated with multimodal imaging, using the radioligand 6-[ 18 F]fluoro-L-DOPA (FDOPA) for positron emission tomography (PET) measurements of dopamine synthesis capacity, and also functional magnetic resonance imaging (fMRI) in a cognitive activation paradigm. We calculated the correlation between FDOPA net blood-brain influx (K app in ; ml/g/min) in the ventral and associative dorsal striatum and BOLD signal changes …

AdultMaleFluorine RadioisotopesDopaminePrefrontal CortexStriatumNucleus accumbensSynaptic TransmissionReward systemmedicineHumansOperculum (brain)Anterior cingulate cortexmedicine.diagnostic_testGeneral NeuroscienceVentral striatumPutamenMiddle AgedMagnetic Resonance ImagingCorpus StriatumDihydroxyphenylalanineDorsolateral prefrontal cortexAffectmedicine.anatomical_structurenervous systemPositron-Emission TomographyCaudate NucleusRadiopharmaceuticalsFunctional magnetic resonance imagingPsychologyNeurosciencepsychological phenomena and processesPhotic StimulationThe European journal of neuroscience
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Reward anticipation revisited- evidence from an fMRI study in euthymic bipolar I patients and healthy first-degree relatives.

2017

Abstract Background Symptomatic phases in bipolar disorder (BD) are hypothesized to result from a hypersensitive behavioral activation system (BAS) being sensitive to potential rewards. However, studies on the neuronal underpinnings of reward anticipation in BD are scarce with contradictory findings and possibly confounded by effects of dopaminergic medication, necessitating further research on dysfunctional motivation in BD. Moreover, its role as vulnerability marker for BD is unclear. Methods Functional imaging was conducted in 16 euthymic BD-I patients free from dopaminergic medication and 19 healthy first-degree relatives using a monetary incentive delay task and compared to parallelize…

AdultMalemedicine.medical_specialtyBipolar DisorderGyrus Cinguli03 medical and health sciences0302 clinical medicineRewardmedicineHumansFamilyBipolar disorderFirst-degree relativesPsychiatryAnterior cingulate cortexMotivationVentral striatumDopaminergicBehavioral activationmedicine.diseaseAnticipationMagnetic Resonance Imaging030227 psychiatryFunctional imagingPsychiatry and Mental healthClinical Psychologymedicine.anatomical_structureFemalePsychologypsychological phenomena and processes030217 neurology & neurosurgeryClinical psychologyJournal of affective disorders
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Childhood methylphenidate treatment of ADHD and response to affective stimuli

2009

Neural correlates of emotional dysregulation in attention-deficit/hyperactivity disorder (ADHD) and persisting influence of Methylphenidate (MPH) still remain insufficiently understood. Decreased activation in the subgenual cingulate and the ventral striatum were found during the perception of positive and negative affective pictures in drug-naïve males with ADHD during childhood (n=10). Males with ADHD during childhood treated with MPH (n=10) did not show any significant differences compared to healthy controls (n=10). Further prospective studies need to clarify direct and indirect mechanisms of MPH treatment that may contribute to emotional processing, which is dysfunctional in males with…

AdultMalemedicine.medical_specialtyEmotionsDysfunctional familyEmotional processingGyrus CinguliBasal Gangliamental disordersmedicineHumansPharmacology (medical)ChildPsychiatryProspective cohort studyBiological PsychiatryPsychiatric Status Rating ScalesPharmacologyNeural correlates of consciousnessMethylphenidateVentral striatumEmotional dysregulationMagnetic Resonance ImagingPsychiatry and Mental healthmedicine.anatomical_structureNeurologyAttention Deficit Disorder with HyperactivityMethylphenidateNeurology (clinical)PsychologyAffective stimuliClinical psychologymedicine.drugEuropean Neuropsychopharmacology
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Opiate-induced dopamine release is modulated by severity of alcohol dependence: an [(18)F]fallypride positron emission tomography study.

2011

Background Preclinical data implicate the reinforcing effects of alcohol to be mediated by interaction between the opioid and dopamine systems of the brain. Specifically, alcohol-induced release of β-endorphins stimulates μ-opioid receptors (MORs), which is believed to cause dopamine release in the brain reward system. Individual differences in opioid or dopamine neurotransmission have been suggested to be responsible for enhanced liability to abuse alcohol. In the present study, a single dose of the MOR agonist remifentanil was administered in detoxified alcohol-dependent patients and healthy control subjects to mimic the β-endorphin-releasing properties of ethanol and to assess the effect…

AgonistAdultMaleFluorine RadioisotopesPyrrolidinesmedicine.drug_classDopamineReceptors Opioid muPharmacologySeverity of Illness IndexRemifentanilRadioligand AssayDopamine receptor D1PiperidinesDopamine receptor D3DopaminemedicineLimbic SystemHumansBiological PsychiatryReceptors Dopamine D2PutamenFunctional NeuroimagingVentral striatumAlcohol dependenceMiddle AgedAnalgesics OpioidBehavior AddictiveAlcoholismmedicine.anatomical_structurenervous systemFallypridePositron-Emission TomographyBenzamidesPsychologymedicine.drugBiological psychiatry
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The Interaction of Person-Affect-Cognition-Execution (I-PACE) model for addictive behaviors: Update, generalization to addictive behaviors beyond int…

2019

We propose an updated version of the Interaction of Person-Affect-Cognition-Execution (I-PACE) model, which we argue to be valid for several types of addictive behaviors, such as gambling, gaming, buying-shopping, and compulsive sexual behavior disorders. Based on recent empirical findings and theoretical considerations, we argue that addictive behaviors develop as a consequence of the interactions between predisposing variables, affective and cognitive responses to specific stimuli, and executive functions, such as inhibitory control and decision-making. In the process of addictive behaviors, the associations between cue-reactivity/craving and diminished inhibitory control contribute to th…

Cognitive Neurosciencemedia_common.quotation_subjectDecision MakingMedizinPrefrontal CortexCravingAffect (psychology)Executive Function03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineddc:150Generalization (learning)mental disordersmedicineHumans0501 psychology and cognitive sciences050102 behavioral science & comparative psychologymedia_commonAddiction05 social sciencesVentral striatumFakultät für Bildungswissenschaften » Institut für Psychologie » Allgemeine Psychologie und SozialpsychologieCognitionModels TheoreticalAmygdalaExecutive functionsBehavior AddictiveInhibition PsychologicalNeuropsychology and Physiological Psychologymedicine.anatomical_structurePsychologieCue reactivityVentral Striatummedicine.symptomPsychology030217 neurology & neurosurgeryCognitive psychologyNeuroscience & Biobehavioral Reviews
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Cross-reinstatement between 3,4-methylenedioxypyrovalerone (MDPV) and cocaine using conditioned place preference.

2019

Abstract 3,4-Methylenedioxypyrovalerone (MDPV) is a new psychoactive substance (NPS) considered to be a cocaine-like psychostimulant. The substitution of an established illicit drug as cocaine with an NPS is a pattern of use reported among drug users. The aim of this study was to investigate the relationship between cocaine and MDPV in the reinstatement of the conditioned place preference (CPP) paradigm, in order to establish whether there is cross-reinstatement between the two psychostimulants. Four experimental groups of male OF1 mice were subjected to the CPP paradigm: MDPV-MDPV, Cocaine-Cocaine, Cocaine-MDPV, and MDPV-Cocaine. The first drug refers to the substance with which the animal…

DrugMaleCannabinoid receptorPyrrolidinesmedia_common.quotation_subjectConditioning ClassicalDrug-Seeking BehaviorMethylenedioxypyrovaleronePharmacology03 medical and health sciencesMice0302 clinical medicineCocaineDopamine Uptake InhibitorsNeuroplasticitymedicineAnimalsBenzodioxolesBiological Psychiatrymedia_commonPharmacologyArc (protein)Dose-Response Relationship Drugbusiness.industryVentral striatumSynthetic CathinoneConditioned place preference030227 psychiatrymedicine.anatomical_structureConditioningbusinessLocomotionmedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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